Human authored, 5 min read – fact checked
High blood pressure (hypertension) is one of the most important modifiable cardiovascular and brain-health risk factors. In England, around 30% of adults aged 16+ had hypertension in 2024, rising to around 70% among those aged 75+ (1), while the WHO estimates that 34% of UK adults aged 30–79 have hypertension, with only around 35% controlled (2). Gibraltar’s GHA data similarly identifies hypertension as a major cardiovascular risk factor; an analysis of 21,448 patient records found 3,564 people with a recorded hypertension diagnosis, although changes in GP coding mean these figures should not be interpreted as a precise population prevalence (2).

Key risk factors for hypertension (3)
– Increasing age and family history
– Excess body weight/central adiposity
– High-salt, highly processed diets
– Physical inactivity
– Excess alcohol consumption
– Smoking
– Chronic stress and poor sleep
– Diabetes/insulin resistance and kidney disease
– Unfavourable lipid profile and broader cardiometabolic risk
Consequences of sustained hypertension (3)
– Increased risk of vascular dementia and cognitive decline
– Increased risk of cardiovascular disease and heart attack
– Increased risk of stroke and small-vessel brain damage
– Increased risk of heart failure`
– Increased risk of chronic kidney disease
– increased risk of damage to blood vessels, eyes and other organs
Client reduces blood pressure
A month after starting her Nutritional Therapy program (fig 2, left), a 66yr old female client reported significant reductions in systolic and diastolic blood pressure, as well as reductions in pulse pressure (the gap between both). Blood pressure was monitored repeatedly, morning and evenings, over several days using the same home blood-pressure monitor, which essentially strengthened the interpretation.
Key findings after a month:
– Systolic blood pressure reduced from 128 to 115 mmHg (−13 mmHg; −10.2%)
– Diastolic blood pressure reduced from 84 to 79 mmHg (−5 mmHg; −6.0%)
– Pulse pressure narrowed by 8 mmHg (~18.2%), reflecting a greater reduction in systolic than diastolic blood pressure.

WHAT IS PULSE PRESSURE?
Pulse pressure is influenced by the interaction between the heart’s stroke volume and the elasticity of the arteries (4). A higher pulse pressure can be a marker of greater arterial stiffness, particularly as people get older. Persistently elevated pulse pressure has also been associated with a greater cardiovascular and cerebrovascular risk. Although pulse pressure alone cannot establish a reduction in arterial stiffness or cardiovascular risk, the outcomes does suggest an encouraging trend in the right direction.
WHY MIGHT ALL THIS MATTER FOR BRAIN HEALTH?
The relationship is particularly important in mid-life. The 2024 Lancet Commission identifies high blood pressure as a modifiable dementia risk factor and reports that persistent mid-life hypertension is associated with greater later-life dementia risk (5). Hypertension may contribute through vascular damage, impaired cerebral blood flow and small-vessel disease, including white-matter changes. Evidence also suggests that effective antihypertensive treatment can reduce the risk of cognitive impairment/dementia, although blood pressure naturally tends to fall in some people approaching dementia, so low BP in later life should not automatically be interpreted as protective.
For context on the second infographic (fig.2, right), I highlighted the outcomes of a recently published study (6), whereby lowering systolic blood pressure to a target under 120 (like the client did), slowed the growth of small blood vessel damage in the brain, compared with the standard target under 140, providing evidence of the broader rationale as to why a sustained lower systolic blood pressure, may be beneficial for brain vascular health. It does not however establish that the client’s blood pressure reduction will prevent vascular dementia.

Working with me to reduce blood pressure
A nutritional therapist can support healthier blood pressure and long-term brain health by identifying and addressing modifiable dietary and lifestyle factors that influence vascular function, metabolic health and inflammation. An individualised approach may include improving overall diet quality, as well as optimising diet, weight and reducing stress. Physical activity is also very improtant. By working with me you will also ensure you are working with a fully qualified PERSONAL TRAINER. Because long-term elevated blood pressure is an established modifiable risk factor for cognitive decline and dementia, improving blood-pressure control through sustainable nutritional and lifestyle changes may also contribute to protecting cerebral blood vessels and reducing the accumulation of vascular brain damage over time. The emphasis is on personalised, measurable and sustainable changes, rather than a one-size-fits-all diet.
- NHS England Digital [Internet]. [cited 2026 Oct 6]. NHS Adults’ health Survey 2024. Available from: https://digital.nhs.uk/data-and-information/publications/statistical/health-survey-for-england/2024/adults-health
- JSNA Cardiovascular Health – Gibraltar Health Authority [Internet]. [cited 2026 Oct 6]. Available from: https://www.gha.gi/jsna/jsna-cardiovascular-health/
- nhs.uk [Internet]. 2024 [cited 2026 Oct 6]. NHS High blood pressure. Available from: https://www.nhs.uk/conditions/high-blood-pressure/
- Cleveland Clinic [Internet]. [cited 2026 Oct 6]. What Is Pulse Pressure? Available from: https://my.clevelandclinic.org/health/body/21629-pulse-pressure
- Livingston G, Huntley J, Liu KY, Costafreda SG, Selbæk G, Alladi S, et al. Dementia prevention, intervention, and care: 2024 report of the Lancet standing Commission. The Lancet. 2024 Aug 10;404(10452):572–628. doi:10.1016/S0140-6736(24)01296-0 PubMed PMID: 39096926.
- Charisis S, Pajewski NM, Price LR, Ho NH, Rashid T, Wang D, et al. Intensive versus standard blood pressure control and overall brain small vessel disease burden: a post-hoc analysis of the SPRINT randomized clinical trial. eClinicalMedicine. 2026 Sep 1;99. doi:10.1016/j.eclinm.2026.104143

